Bispecific antisense oligonucleotides that inhibit IGFBP-2 and IGFBP-5 and methods of using same
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Bispecific antisense oligonucleotides which consist essentially of a sequence of bases that is complementary to portions of both the gene encoding human IGFBP-2 and the gene encoding human IGFBP-5 are useful in as antisense therapeutics in the treatment of endocrine-regulated cancers.