Alzheimer’s Disease (AD) comprise the majority of late-onset dementias. AD affects the brain, resulting in loss of memory, decision making, and motor function. In the next 15 years, AD, along with cancer and heart diseases, will become a significant socioeconomic burden that needs to be addressed. AD is a progressive neurodegenerative disorder that doubles in chance every 5 years for individuals above the age of 60. This chance increases to 10% above 65, and near 50% for 85+ individuals. The main causes for AD are primarily genetic (inheriting the e4 APOE gene), with brain damage a known secondary associate. Recent research posits that infection of the herpes simplex type 1 or chlamydia virus results in an AD phenotype. These factors result in the death and weakening of neurons, which is only worse for the elderly as they age, and the effects of radical oxygen species seem to exacerbate this phenomenon. Coriandrum sativum, along with Umbelliferae are known medicinal herbs - most commonly used for helping with digestion. Futhermore, previous studies indicate the anti-inflammatory effects of C. sativum. This study aims to investigate the anti-inflammatory, anti-radical species effects of C. sativum to combat AD. In in vitro, SH-SY6Y cells were used to determine the efficacy of C. sativum on neuronal protection. In vivo tests were conducted using Alzheimer’s Disease Drosophila models. C. sativum’s effects on cell death, anti-inflammation, ROS degradation were tested.