Large, myelinated, motor A-fibers in the vagus innervate muscles of the larynx. They are implicated in side effects of cervical vagus nerve stimulation (VNS), laryngeal paralysis after surgical injury of the vagus, and in motor manifestations of progressive bulbar palsy. Knowledge of the degree of engagement of A-fibers by vagal stimuli is desirable for optimization of VNS parameters and for functional assessment of the motor neuron-laryngeal muscle projection. In mice, the most widely used animal model in preclinical studies, due to the short length of the vagus and the fast conduction velocity of A-fibers, direct measurement of stimulus-evoked A-fiber potentials is not feasible. Here, we describe a method to estimate vagal A-fiber engagement in mice by measuring stimulus-evoked EMG (eEMG) from the thyroarytenoid-lateral cricoarytenoid laryngeal muscle complex using intramuscular wires. Recorded eEMG was typically biphasic, with 1.87-2.68 ms latency and 1.05-1.6 ms duration. Stimulus intensity threshold (T) for eEMG was 2.6-24uA (100us square pulses), lower than that for heart rate changes, which indicate engagement of B-fibers (3-7xT; $10.4-72\ \ \mu \mathrm{A}$), and for breathing changes, which indicate engagement of C-fibers (9-25xT; $65-216\ \mu \mathrm{A}$). eEMG amplitude increased with stimulus intensity and saturated at around 7-10xT ($20-65\ \mu \mathrm{A}$). Vagotomy caudal to the stimulating electrode and rostral to the recurrent laryngeal nerve resulted in disappearance of the eEMG.