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요약정보
Ang Ⅱ诱导大鼠成肌细胞萎缩模型的构建 / Establishment of Rat Myoblast Atrophy Model Induced by Ang Ⅱ
Resource Type
Academic Journal
Authors
宿玮洁
;
张美沙
;
李佳凝
;
刘颖
;
徐丁洁
;
常宏
;
SU Wei-jie
;
ZHANG Mei-sha
;
LI Jia-ning
;
LIU Ying
;
XU Ding-jie
;
CHANG Hong
Source
昆明医科大学学报 / Journal of Kunming Medical University
. 42(10):8-13
Subject
L6细胞;心力衰竭;肌少症;骨骼肌萎缩模型
Language
Chinese
ISSN
1003-4706
Online Access
Find it @ DONGA
초록
目的 构建Ang Ⅱ诱导大鼠成肌细胞(L6)萎缩模型,为进一步研究心衰合并肌少症提供模型依据.方法 采用大鼠成肌细胞(L6),用2%马血清诱导分化,镜下观察和MyHC蛋白表达情况判断诱导分化的时间.CCK-8检测不同浓度Ang Ⅱ对L6细胞的影响.为确定细胞萎缩模型的条件,选择不同浓度的Ang Ⅱ作用于L6细胞,并用Western Blot和免疫荧光检测MyHC蛋白的表达情况.结果 L6细胞在2%马血清下生长6d,能被成功诱导分化,镜下显示细胞出现融合现象和肌管,MyHC蛋白表达也增加.0.1~10μM的Ang Ⅱ对L6细胞活性无影响,而Western Blot和免疫荧光检测提示在5 μM和10μM的Ang Ⅱ作用72 h下,MyHC蛋白表达水平逐渐下降.结论 诱导分化后的L6细胞经10 μM Ang Ⅱ刺激72 h可形成细胞萎缩模型,为心衰合并肌少症的体外细胞研究提供基础.
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