Involuntary, forced or voluntary exercise can ameliorate the cognitive deficits by enhancing levels of hippocampal NMDAR1, pAMPAR1 and pCaMKII in a model of vascular dementia
- Resource Type
- Authors
- Zunlin Gao; Suiying Peng; Peihui Wu; Longfei You; Yangyang Lin; Yangfan Xu; Huiting Feng; Juntao Dong; Yujie Deng
- Source
- Neurological Research. 43:349-357
- Subject
- Male
0301 basic medicine
Neurogenesis
Hippocampus
Motor Activity
Hippocampal formation
Receptors, N-Methyl-D-Aspartate
03 medical and health sciences
0302 clinical medicine
Cog
Morris Water Maze Test
Physical Conditioning, Animal
mental disorders
Animals
Medicine
Functional electrical stimulation
Cognitive Dysfunction
Receptors, AMPA
Cognitive skill
Rats, Wistar
Vascular dementia
bcl-2-Associated X Protein
business.industry
Dementia, Vascular
Cognition
Long-term potentiation
General Medicine
medicine.disease
Electric Stimulation
Rats
Disease Models, Animal
030104 developmental biology
Neurology
bcl-Associated Death Protein
Neurology (clinical)
Calcium-Calmodulin-Dependent Protein Kinase Type 2
business
human activities
Neuroscience
030217 neurology & neurosurgery
- Language
- ISSN
- 1743-1328
0161-6412
Objectives: To investigate the effect on vascular dementia of involuntary exercise induced by functional electrical stimulation and of forced and voluntary exercise, focusing on the recovery of cog...