The prevalent I686T human variant and loss-of-function mutations in the cardiomyocyte-specific kinase gene TNNI3K cause adverse contractility and concentric remodeling in mice.
- Resource Type
- Article
- Source
- Human Molecular Genetics; Nov2020, Vol. 29 Issue 21, p3504-3515, 12p
- Subject
- Language
- ISSN
- 09646906